Ling Yin, Speaker at Renewable Energy Conferences
Weifang Institute of Technology, China
Title : Synergistic antifibrotic potential of protocatechuic acid and D-carvone in liver protection

Abstract:

Background: Liver fibrosis, driven by chronic injury, oxidative stress, and inflammation, represents a significant global health burden. Natural compounds such as protocatechuic acid (PCA) and D-Carvone (D-Car) possess known antioxidant and anti-inflammatory properties, but their combined therapeutic potential against liver fibrosis remains unexplored.

Purpose: This study aimed to investigate the synergistic antifibrotic effects of PCA and D-Car in both cellular and animal models of liver fibrosis.

Methods: A combined in vitro and in vivo approach was employed. In vitro experiments utilized the HSC-T6 hepatic stellate cell line, where cells were treated with PCA (50 μM) and D-Car (50 ng/mL), individually and in combination. Cytotoxicity (MTT assay), apoptosis (flow cytometry and Hoechst staining), and Annexin A2 levels were assessed. In vivo studies involved a CCl₄-induced liver fibrosis rat model. Following treatment with PCA, D-Car, or their combination, body weight, liver enzymes (ALT, AST, ALP, albumin), LDH activity, fibrotic markers (TIMP-1, Col1α1), antioxidant enzymes (SOD, Cyp2e1), inflammatory cytokines (IL-6, TNF-α, NF-κB), and histological changes were evaluated.

Results: PCA+D-Car treatment significantly improved HSC-T6 cell morphology, reduced cytotoxicity (*p* < 0.001), and decreased apoptosis compared to individual treatments. In vivo, PCA+D-Car restored body weight, normalized liver enzymes, downregulated TIMP-1 and Col1α1, enhanced antioxidant activity (SOD1), and reduced inflammatory markers (*p* < 0.01). Histological analysis confirmed improved liver architecture with reduced fibrosis and inflammation.

Conclusion: The combination of PCA and D-Carvone exhibits synergistic antifibrotic effects by mitigating oxidative stress, inflammation, and extracellular matrix deposition. These findings highlight its potential as a dietary or therapeutic strategy for liver fibrosis, warranting further clinical exploration.
 

Biography:

Ling Yin is a researcher affiliated with the College of Medicine at the University of Florida, USA. Her research interests focus on hepatology, particularly the molecular mechanisms of liver fibrosis and the development of novel therapeutic strategies using natural compounds. She has contributed to several studies investigating the role of oxidative stress and inflammation in hepatic pathogenesis and the protective effects of phytochemicals.

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