HYBRID EVENT: You can participate in person at Tokyo, Japan from your home or work.
Cancer Autophagy a cellular process crucial for maintaining cellular homeostasis, has emerged as a significant player in the context of cancer. Autophagy, often referred to as "self-eating," involves the degradation and recycling of cellular components. While it is a fundamental process for normal cell function, dysregulation of autophagy has been implicated in various diseases, including cancer. In the realm of cancer biology, autophagy plays a dual role, acting both as a tumor suppressor and a pro-survival mechanism. On one hand, it functions to eliminate damaged organelles and proteins, preventing the accumulation of harmful cellular material that could lead to genomic instability—a hallmark of cancer. On the other hand, cancer cells exploit autophagy to endure stressful conditions, such as nutrient deprivation or hypoxia, promoting their survival and resistance to therapy. Understanding the intricate relationship between cancer and autophagy is pivotal for developing targeted therapeutic strategies. Researchers are actively exploring ways to manipulate autophagy to enhance the efficacy of cancer treatments. Modulating autophagy presents a promising avenue for sensitizing cancer cells to conventional therapies or even as a standalone therapeutic approach. Moreover, the complex interplay between autophagy and various signaling pathways within cancer cells adds another layer of intricacy to this field of study. Unraveling these molecular mechanisms holds the key to unlocking novel therapeutic targets and advancing our ability to combat cancer.
In conclusion, the study of cancer autophagy continues to be a dynamic and evolving field within cancer research. As we delve deeper into the molecular intricacies of this process, new opportunities for therapeutic intervention are likely to emerge, offering hope for more effective cancer treatments in the future.
